North American dominance

A BREAK FROM 

North American dominance 

--------------------------------------------------------------------

We expect like while in the Mediterranean outside of Canada & USA social context not to rely solely around North American media, content & branding plus Government's + history 

Topics brought up & no reference to like in North America where is is very focused & dominant over archives of documented other works, events & interest in earth's history 

Everything 1920's and in North America all Hollywood specific modern content is referred to as the only subject yet... 

There are many other options alongside or separate 

H.I.3 Case

The three clusters of Ontario labs are guilty of this regularly especially between 2012-2023

Havana International 3+

The North Americans ----------------------------------

Attempts to force us into sterilization then disability - handicap claims to cover up wireless imprisonment while efforts to convert us to being LGBTQ2+ Lesbian, gay, bisexual, transgender & queer in wirelessly manufactured, isolated simple lives holding us accountable to false claims & what is forced upon is against our will then attacking us for it 

We are 100% against Canada & those in USA or internationally that operated with & at the three clusters of Ontario labs 

--------------------------------------------------------------------

--------------------------------------------------------------------

WE DO NOT WANT THE CANADIAN'S ON EARTH

Those that are fine being screened for another country are welcome to after they are educated 

The remainder are not welcome over H.I.3

Canada should exit UN United Nations & Commonwealth Org as a member countries 

The Canadians expect to own everyone with US & Mexican interests as wireless full body Havana Syndrome causing drone robot puppet ventriloquist doll technology 

We are to be so stupid that we will not understand or know what to do & be forced to accept & just die early while all signs of our existence is destroyed & we are forgotten like we never existed 

Canada is not welcome as a Nato member country. Canada cannot be trusted 

We are destroying Canada in every area of every country over the 2012-2023 rape-torture-terror attacks on Dr Sydney Bennett & Dr Carly Koslov + efforts taken prior to 2012

Canada is decided. The United States is devided 

FUTURE OF FILM - TELEVISION -------------------

Main careers

Focus on characters

Are big names going to exist as they had dominated in "American" focus entertainment

Same small group & thousands of films & television series produced

NAPALM & CYNADE

Napalm is a weaponized mixture of chemicals designed to create a highly flammable and gelatinous liquid. The initial thickening agent was a combination of naphthenic and palmitic acids leading to the trade name “na-palm” but more generically known as firebomb fuel-gel mixture

Many variations of the chemicals used in napalm exist. The most common current composition includes aluminum salts, polystyrene, and benzene. Detonation then occurs by various explosive compounds that ignite phosphorous, which burns at a temperature adequate to ignite the fuel mixture

The creation of napalm (1942): the invention of an “efficient” incendiary weapon. The creation of napalm on 4 July 1942 by Louis Fieser crowned a succession of experiments on the Harvard campus beginning in 1940 under the direction of the National Defense Research Committee

Napalm is an incendiary mixture of a gelling agent and a volatile petrochemical (usually petrol or diesel fuel). The name is a portmanteau of two of the constituents of the original thickening and gelling agents: coprecipitated aluminium salts of naphthenic acid and palmitic acid. Napalm B is the more modern version of napalm (utilizing polystyrene derivatives) and, although distinctly different in its chemical composition, is often referred to simply as "napalm". A team led by chemist Louis Fieser originally developed napalm for the US Chemical Warfare Service in 1942 in a secret laboratory at Harvard University. Of immediate first interest was its viability as an incendiary device to be used in fire bombing campaigns during World War II; its potential to be coherently projected into a solid stream that would carry for distance (instead of the bloomy fireball of pure gasoline) resulted in widespread adoption in infantry flamethrowers as well

Napalm burns at temperatures ranging from 800 to 1,200 °C (1,470 to 2,190 °F). It burns longer than gasoline, is more easily dispersed, and adheres to its targets. These traits make it effective and controversial. It has been widely deployed from the air and from the ground, the largest use being via airdropped bombs in World War II in the incendiary attacks on Japanese cities in 1945. It was used also for close air support roles in the First Indochina War, the Algerian War, the Korean War and the Second Indochina War. Napalm has also fueled most of the flamethrowers (tank-, ship-, and infantry-based) used since World War II, giving them much greater range, and was a common weapon of urban combat by both the Axis and the Allies in World War II

Napalm was used in flamethrowers, bombs, and tanks in World War II. It is believed to have been formulated to burn at a specific rate and to adhere to surfaces to increase its stopping power. During combustion, napalm rapidly deoxygenates the available air and generates large amounts of carbon monoxide and carbon dioxide

Alternative compositions exist for different types of uses, e.g., triethylaluminium, a pyrophoric compound that aids ignition

Use of fire in warfare has a long history. Greek fire, which also is described as "sticky fire" (πῦρ κολλητικόν, pýr kolletikón), is believed to have had a petroleum base. The development of napalm was precipitated by the use of jellied gasoline mixtures by the Allied forces during World War II.The use of aluminium salts of organic acids (Ionov's salt) for the preparation of incendiary viscous mixtures was already done by the Soviets in 1939, with high acceptance by the Red Army. Latex, used in these early forms of incendiary devices, became scarce, since natural rubber was almost impossible to obtain after the Japanese army captured the rubber plantations in Malaya, Indonesia, Vietnam, and Thailand.

This shortage of natural rubber prompted chemists at US companies such as DuPont and Standard Oil of New Jersey, and researchers at Harvard University, to develop factory-made alternatives—artificial rubber for all uses, including vehicle tires, tank tracks, gaskets, hoses, medical supplies and rain clothing. A team of chemists led by Louis Fieser at Harvard University was the first to develop synthetic napalm during 1942. "The production of napalm was first entrusted to Nuodex Products, and by the middle of April 1942 they had developed a brown, dry powder that was not sticky by itself, but when mixed with gasoline turned into an extremely sticky and flammable substance." One of Fieser's colleagues suggested adding phosphorus to the mix which increased the "ability to penetrate deeply [...] into the musculature, where it would continue to burn day after day."

On 4 July 1942, the first test occurred on the football field near the Harvard Business School. Tests under operational conditions were carried out at Jefferson Proving Ground on condemned farm buildings and subsequently at Dugway Proving Ground on buildings designed and constructed to represent those to be found in German and Japanese towns. This new mixture of chemicals was widely used by the United States in the Second World War for incendiary bombs and in flamethrowers, after its first deployment in Papua New Guinea on 15 December 1943.

From 1965 to 1969, the Dow Chemical Company manufactured napalm B for the American armed forces. After news reports of napalm B's deadly and disfiguring effects were published, Dow Chemical experienced boycotts of its products, and its recruiters for new chemists, chemical engineers, etc., graduating from college were subject to campus boycotts and protests. The management of the company decided that its "first obligation was the government". Meanwhile, napalm B became a symbol for the Vietnam War.

When used as a part of an incendiary weapon, napalm can cause severe burns (ranging from superficial to subdermal), asphyxiation, unconsciousness, and death. In this implementation, napalm fires can create an atmosphere of greater than 20% carbon monoxide and firestorms with self-perpetuating winds of up to 110 kilometers per hour (70 mph).

Napalm is effective against dug-in enemy personnel. The burning incendiary composition flows into foxholes, tunnels, and bunkers, and drainage and irrigation ditches and other improvised troop shelters. Even people in undamaged shelters can be killed by hyperthermia, radiant heat, dehydration, asphyxiation, smoke exposure, or carbon monoxide poisoning. Crews of armored fighting vehicles are also vulnerable, due to the intense heat conducted through the armor. Even in the case of a near miss, the heat can be enough to disable a vehicle.

One firebomb released from a low-flying plane can damage an area of 2,100 square meters (2,500 sq yd).

How Napalm Works 

https://science.howstuffworks.com/napalm.htm

In chemistry, a cyanide is a chemical compound that contains a C≡N functional group. This group, known as the cyano group, consists of a carbon atom triple-bonded to a nitrogen atom. In inorganic cyanides, the cyanide group is present as the cyanide anion ⁻C≡N. This anion is extremely poisonous. 

Formula: CN−

Molar mass: 26.02 g/mol

3D model (JSmol): Interactive image

Conjugate acid: Hydrogen cyanide

PubChem CID: 5975

The above-given compound is Cyanide (CN⁻). The carbon is bonded to the nitrogen atom with triple bonds, and the charge of the cyanide atom is -1.

Cyanides are chemical compounds which are fast acting poisons with a chemical formula CN−. Cyanides consist of one nitrogen atom and a carbon atom connected to each other with a triple bond. It is also known as cyanide anion or nitrile anion, cyanide ions. Certain algae, fungi, and bacteria can produce cyanide.

The word "cyanide" comes from the Greek word for "blue", in reference to HCN, which was called Blausäure ("blue acid") in German after its preparation by acid treatment of Prussian blue.

In industry, it is used in manufacturing, pesticides, and can be found in several industrial chemicals. 2,3 In medicine, cyanide can be found in the widely used antihypertensive sodium nitroprusside, each molecule of which contains 5 ions of cyanide.

In large doses, the body's ability to change cyanide into thiocyanate is overwhelmed. Large doses of cyanide prevent cells from using oxygen and eventually these cells die. The heart, respiratory system and central nervous system are most susceptible to cyanide poisoning.

You can also find cyanide in certain nitrile compounds used in medications like citalopram (Celexa) and cimetidine (Tagamet). Nitriles aren't as toxic because they don't easily release the carbon-nitrogen ion, which is what acts as a poison in the body. Cyanide is even a byproduct of metabolism in the human body.

Levels of 0.5–1 mg/L are mild, 1–2 mg/L are moderate, 2–3 mg/L are severe, and greater than 3 mg/L generally result in death. If exposure is suspected, the person should be removed from the source of the exposure and decontaminated. Treatment involves supportive care and giving the person 100% oxygen.

Low levels of cyanides are found in living organisms and are present in cigarette smoke, vehicle exhaust, and in foods such as spinach, bamboo shoots, almonds, lima beans, fruit pits and tapioca.

It is rapidly absorbed, symptoms begin few seconds after exposure and death usually occurs in <30 min. The average lethal dose for potassium cyanide is about 250 mg.

A single cherry contains 0.17 grams of cyanide per each gram of seed. So, depending on the size of the pit, it will take a lot of freshly-ground cherry seeds (approximately 588) to give you a lethal dose. So, swallowing one or two of them accidentally won't kill you.

Cherry pits contain trace amounts of cyanide. While accidentally eating a few cherry pits will not kill you, eating more than 20-30 can lead to dangerous toxicity. Your body can withstand up to 50 mg of cyanide before it becomes poisonous.

Cassava, sorghum, stone fruits, bamboo roots and almonds are especially important foods containing cyanogenic glycosides. The potential toxicity of a cyanogenic plant depends primarily on the potential that its consumption will produce a concentration of cyanide that is toxic to exposed humans.

However, because of cyanide's high acute toxicity, accidental spills have caused mass mortality events of aquatic life and pose an acute human health risk where water affected by the spill is used as a drinking water supply.”

Both arsenic and cyanide are poisonous chemical materials. The key difference between arsenic and cyanide is that arsenic is a poisonous chemical element that occurs as a metalloid, whereas cyanide is a group of poisonous chemical compounds consisting of a cyanide functional group.

Bitter almonds are those that naturally contain a toxin that your body breaks down into cyanide — a compound that can cause poisoning and even death. For this reason, raw bitter almonds should not be eaten. Boiling, roasting, or microwaving bitter almonds may help reduce their toxin content and make them safer to eat.

Cyanide use in mining

Gold is one of the noble metals and as such it is not soluble in water. Cyanide, which stabilizes the gold species in solution, and an oxidant such as oxygen are required to dissolve gold. The first step in the process is to prepare the ore by crushing and grinding.

Lima beans

Like many legumes, the seemingly innocent lima bean should not be eaten raw — doing so can be lethal. (And who wants to die in such an ignoble way as death by lima bean?) Also known as butter beans, the legumes can contain a high level of cyanide, which is part of the plant's defense mechanism.

Fruit and vegetables that produce cyanide

These fruits include apricots, cherries, peaches, pears, plums and prunes. The flesh of the fruit itself is not toxic.

To start, stick to one serving (1/2 cup or about 7 cherries, depending on their size), see how your gut reacts, and go from there. Take the time to measure them out, so you're not tempted to keep popping them in your mouth—otherwise, you may risk your insides retaliating.

Maraschino cherries, also known as cocktail cherries, are preserved, sweetened, and artificially colored to become bright red cherries. That's why they have a different taste and composition (no pits) than natural cherries.

Cyanide poisoning is poisoning that results from exposure to any of a number of forms of cyanide. Early symptoms include headache, dizziness, fast heart rate, shortness of breath, and vomiting. This phase may then be followed by seizures, slow heart rate, low blood pressure, loss of consciousness, and cardiac arrest. Onset of symptoms usually occurs within a few minutes. Some survivors have long-term neurological problems.

Toxic cyanide-containing compounds include hydrogen cyanide gas and a number of cyanide salts. Poisoning is relatively common following breathing in smoke from a house fire. Other potential routes of exposure include workplaces involved in metal polishing, certain insecticides, the medication sodium nitroprusside, and certain seeds such as those of apples and apricots. Liquid forms of cyanide can be absorbed through the skin. Cyanide ions interfere with cellular respiration, resulting in the body's tissues being unable to use oxygen.

Diagnosis is often difficult. It may be suspected in a person following a house fire who has a decreased level of consciousness, low blood pressure, or high lactic acid. Blood levels of cyanide can be measured but take time. Levels of 0.5–1 mg/L are mild, 1–2 mg/L are moderate, 2–3 mg/L are severe, and greater than 3 mg/L generally result in death.

If exposure is suspected, the person should be removed from the source of the exposure and decontaminated. Treatment involves supportive care and giving the person 100% oxygen. Hydroxocobalamin (vitamin B12a) appears to be useful as an antidote and is generally first-line. Sodium thiosulphate may also be given. Historically, cyanide has been used for mass suicide and it was used for genocide by the Nazis.

If hydrogen cyanide is inhaled, it can cause a coma with seizures, apnea, and cardiac arrest, with death following in a matter of seconds. At lower doses, loss of consciousness may be preceded by general weakness, dizziness, headaches, vertigo, confusion, and perceived difficulty in breathing. At the first stages of unconsciousness, breathing is often sufficient or even rapid, although the state of the person progresses towards a deep coma, sometimes accompanied by pulmonary edema, and finally cardiac arrest. A cherry red skin color that darkens may be present as the result of increased venous hemoglobin oxygen saturation. Despite the similar name, cyanide does not directly cause cyanosis. A fatal dose for humans can be as low as 1.5 mg/kg body weight. Other sources claim a lethal dose is 1–3 mg per kg body weight for vertebrates.

Exposure to lower levels of cyanide over a long period (e.g., after use of improperly processed cassava roots, which are a primary food source in tropical Africa) results in increased blood cyanide levels, which can result in weakness and a variety of symptoms, including permanent paralysis, nervous lesions, hypothyroidism, and miscarriages. Other effects include mild liver and kidney damage.

Cyanide poisoning can result from the ingestion of cyanide salts; imbibing pure liquid prussic acid; skin absorption of prussic acid; intravenous infusion of nitroprusside for hypertensive crisis; or the inhalation of hydrogen cyanide gas. The last typically occurs through one of three mechanisms:

The gas is directly released from canisters (e.g. as part of a pesticide, insecticide, or Zyklon B).It is generated on site by reacting potassium cyanide or sodium cyanide with sulfuric acid (e.g. in a modern American gas chamber).Fumes arise during a building fire or any similar scenario involving the burning of polyurethane, vinyl or other polymer products that required nitriles in their production.

As potential contributing factors, cyanide is present in:

Tobacco smoke.Many seeds or kernels such as those of almonds, apricots, apples, oranges, and flaxseed. Foods including cassava (also known as tapioca, yuca or manioc) and bamboo shoots.

As a potential harm-reduction factor, Vitamin B12, in the form of hydroxocobalamin (also spelled hydroxycobalamin), might reduce the negative effects of chronic exposure; whereas, a deficiency might worsen negative health effects following exposure to cyanide.

Cyanide is a potent cytochrome c oxidase (COX, a.k.a. Complex IV) inhibitor. As such, cyanide poisoning is a form of histotoxic hypoxia, because it interferes with oxidative phosphorylation.

Specifically, cyanide binds to the heme a3-CuB binuclear center of COX (and thus is a non-competitive inhibitor of it). This prevents electrons passing through COX from being transferred to O2, which not only blocks the mitochondrial electron transport chain, it also interferes with the pumping of a proton out of the mitochondrial matrix which would otherwise occur at this stage. Therefore, cyanide interferes not only with aerobic respiration but also with the ATP synthesis pathway it facilitates, owing to the close relationship between those two processes.

One antidote for cyanide poisoning, nitrite (i.e. via amyl nitrite), works by converting ferrohemoglobin to ferrihemoglobin, which can then compete with COX for free cyanide (as the cyanide will bind to the iron in its heme groups instead). Ferrihemoglobin cannot carry oxygen, but the amount of ferrihemoglobin that can be formed without impairing oxygen transport is much greater than the amount of COX in the body.

Cyanide is a broad-spectrum poison because the reaction it inhibits is essential to aerobic metabolism; COX is found in many forms of life. However, susceptibility to cyanide is far from uniform across affected species; for instance, plants have an alternative electron transfer pathway available that passes electrons directly from ubiquinone to O2, which confers cyanide resistance by bypassing COX.

Lactate is produced by anaerobic glycolysis when oxygen concentration becomes too low for the normal aerobic respiration pathway. Cyanide poisoning inhibits aerobic respiration and therefore increases anaerobic glycolysis which causes a rise of lactate in the plasma. A lactate concentration above 10 mmol per liter is an indicator of cyanide poisoning, as defined by the presence of a blood cyanide concentration above 40 µmol per liter. Lactate levels greater than 6 mmol/L after reported or strongly suspected pure cyanide poisoning, such as cyanide-containing smoke exposure, suggests significant cyanide exposure.

Methods of detection include colorimetric assays such as the Prussian blue test, the pyridine-barbiturate assay, also known as the "Conway diffusion method" and the taurine fluorescence-HPLC but like all colorimetric assays these are prone to false positives. Lipid peroxidation resulting in "TBARS," an artifact of heart attack produces dialdehydes that cross-react with the pyridine-barbiturate assay. Meanwhile, the taurine-fluorescence-HPLC assay used for cyanide detection is identical to the assay used to detect glutathione in spinal fluid.

Cyanide and thiocyanate assays have been run with mass spectrometry (LC/MS/MS), which are considered specific tests. Since cyanide has a short half-life, the main metabolite, thiocyanate is typically measured to determine exposure. Other methods of detection include the identification of plasma lactate.

Decontamination

Decontamination of people exposed to hydrogen cyanide gas only requires removal of the outer clothing and the washing of their hair: Those exposed to liquids or powders generally require full decontamination.

Antidote

The International Programme on Chemical Safety issued a survey (IPCS/CEC Evaluation of Antidotes Series) that lists the following antidotal agents and their effects: oxygen, sodium thiosulfate, amyl nitrite, sodium nitrite, 4-dimethylaminophenol, hydroxocobalamin, and dicobalt edetate ('Kelocyanor'), as well as several others. Other commonly-recommended antidotes are 'solutions A and B' (a solution of ferrous sulfate in aqueous citric acid, and aqueous sodium carbonate, respectively) and amyl nitrite.

The United States standard cyanide antidote kit first uses a small inhaled dose of amyl nitrite, followed by intravenous sodium nitrite, followed by intravenous sodium thiosulfate. Hydroxocobalamin was approved for use in the US in late 2006 and is available in Cyanokit antidote kits. Sulfanegen TEA, which could be delivered to the body through an intra-muscular (IM) injection, detoxifies cyanide and converts the cyanide into thiocyanate, a less toxic substance. Alternative methods of treating cyanide intoxication are used in other countries.

The Irish Health and Safety Executive (HSE) has recommended against the use of solutions A and B because of their limited shelf life, potential to cause iron poisoning, and limited applicability (effective only in cases of cyanide ingestion, whereas the main modes of poisoning are inhalation and skin contact). The HSE has also questioned the usefulness of amyl nitrite due to storage/availability problems, risk of abuse, and lack of evidence of significant benefits. It also states that the availability of kelocyanor at the workplace may mislead doctors into treating a patient for cyanide poisoning when this is an erroneous diagnosis. The HSE no longer recommends a particular cyanide antidote.

Fires

The República Cromañón nightclub fire broke out in Buenos Aires, Argentina on 30 December 2004, killing 194 people and leaving at least 1,492 injured. Most of the victims died from inhaling poisonous gases, and carbon monoxide. After the fire, the technical institution INTI found that the level of toxicity due to the materials and volume of the building was 225 ppm of cyanide in the air. A lethal dose for rats is between 150 ppm and 220 ppm, meaning the air in the building was highly toxic.

On 5 December 2009, a fire in the night club Lame Horse (Khromaya Loshad) in the Russian city of Perm took the lives of 156 people. Fatalities consisted of 111 people at the site and 45 later in hospitals. One of the main causes of death was poisoning from cyanide and other toxic gases released by the burning of plastic and polyurethane foam used in the construction of club interiors. Taking into account the number of deaths, this was the largest fire in post-Soviet Russia.

On 27 January 2013, a fire at the Kiss nightclub in the city of Santa Maria, in the south of Brazil, caused the poisoning of hundreds of young people by cyanide released by the combustion of soundproofing foam made with polyurethane. By March 2013, 245 fatalities were confirmed.

Research of hydrogen cyanide by chemists Carl Wilhelm Scheele and Claude Bernard would become central to understanding the lethality of future gas chambers. In early 1942, Zyklon B, which contains hydrogen cyanide, emerged as the preferred killing tool of Nazi Germany for use in extermination camps during the Holocaust.The chemical was used to murder roughly one million people in gas chambers installed in extermination camps at Auschwitz-Birkenau, Majdanek, and elsewhere. Most of the people who were murdered were Jews, and by far the majority of these murders took place at Auschwitz. Zyklon B was supplied to concentration camps at Mauthausen, Dachau, and Buchenwald by the distributor Heli, and to Auschwitz and Majdanek by Testa. Camps also occasionally bought Zyklon B directly from the manufacturers. Of the 729 tonnes of Zyklon B sold in Germany in 1942–44, 56 tonnes (about eight percent of domestic sales) were sold to concentration camps. Auschwitz received 23.8 tonnes, of which six tonnes were used for fumigation. The remainder was used in the gas chambers or lost to spoilage (the product had a stated shelf life of only three months). Testa conducted fumigations for the Wehrmacht and supplied them with Zyklon B. They also offered courses to the SS in the safe handling and use of the material for fumigation purposes. In April 1941, the German agriculture and interior ministries designated the SS as an authorized applier of the chemical, and thus they were able to use it without any further training or governmental oversight.

Hydrogen cyanide as has been used for judicial execution in some states of the United States, where cyanide was generated by reaction between potassium cyanide (or sodium cyanide) dropped into a compartment containing sulfuric acid, directly below the chair in the gas chamber.

Suicide or Murder covered up as overdose or

Cyanide salts are sometimes used as fast-acting suicide devices. Cyanide reacts at a higher level with high stomach acidity.

On 26 January 1904, company promoter and swindler Whitaker Wright died by suicide by ingesting cyanide in a court anteroom immediately after being convicted of fraud.In February 1937, the Uruguayan short story writer Horacio Quiroga died by suicide by drinking cyanide in a hospital at Buenos Aires.In 1937, polymer chemist Wallace Carothers died by suicide by cyanide.In the 1943 Operation Gunnerside to destroy the Vemork Heavy Water Plant in World War II (an attempt to stop or slow German atomic bomb progress), the commandos were given cyanide tablets (cyanide enclosed in rubber) kept in the mouth and were instructed to bite into them in case of German capture. The tablets ensured death within three minutes. Cyanide, in the form of pure liquid prussic acid (a historical name for hydrogen cyanide), was the favored suicide agent of Nazi Germany. Erwin Rommel (1944), Adolf Hitler's wife, Eva Braun (1945), and Nazi leaders Heinrich Himmler (1945), possibly Martin Bormann (1945), and Hermann Göring (1946) all died by suicide by ingesting it.It is speculated that, in 1954, Alan Turing used an apple that had been injected with a solution of cyanide to die by suicide after being convicted of having a homosexual relationship, which was illegal at the time in the United Kingdom, and forced to undergo hormonal castration to avoid prison. An inquest determined that Turing's death from cyanide poisoning was a suicide, although this has been disputed.Adolf Hitler's wife, Eva Braun, committed suicide via hydrogen cyanide in the Führerbunker on April 30th, 1945. Members of the Sri Lankan Tamil (or Eelam Tamil) LTTE (Liberation Tigers of Tamil Eelam, whose insurgency lasted from 1983 to 2009), used to wear cyanide vials around their necks with the intention of dying by suicide if captured by the government forces.On 22 June 1977, Moscow, Aleksandr Dmitrievich Ogorodnik, a Soviet diplomat accused of spying on behalf of the Colombian Intelligence Agency and the US Central Intelligence Agency, was arrested. During the interrogations, Ogorodnik offered to write a full confession and asked for his pen. Inside the pen cap was a cleverly hidden cyanide pill, which when bitten on, caused Ogorodnik to die before he hit the floor, according to the Soviets. On 18 November 1978, Jonestown. A total of 909 individuals died in Jonestown, many from apparent cyanide poisoning, in an event termed "revolutionary suicide" by Jones and some members on an audio tape of the event and in prior discussions. The poisonings in Jonestown followed the murder of five others by Temple members at Port Kaituma, including United States Congressman Leo Ryan, an act that Jones ordered. Four other Temple members died by murder-suicide in Georgetown at Jones' command.On 6 June 1985, serial killer Leonard Lake died in custody after having ingested cyanide pills he had sewn into his clothes.On 28 June 2012, Wall Street trader Michael Marin ingested a cyanide pill seconds after a guilty verdict was read in his arson trial in Phoenix, AZ; he died minutes after. On 22 June 2015, John B. McLemore, a horologist and the central figure of the podcast S-Town, died after ingesting cyanide. On 29 November 2017, Slobodan Praljak died from drinking potassium cyanide, after being convicted of war crimes by the International Criminal Tribunal for the former Yugoslavia.

Murder

John Tawell, a murderer who in 1845 became the first person to be arrested as the result of telecommunications technology.Grigori Rasputin (1916; attempted, later killed by gunshot)The Goebbels children (1945)

Stepan Bandera (1959)

Jonestown, Guyana, was the site of a large mass murder–suicide in which over 900 members of the Peoples Temple drank potassium cyanide–laced Flavor Aid in 1978.Chicago Tylenol murders (1982)

Timothy Marc O'Bryan (1966–1974) died on October 31, 1974 by ingesting potassium cyanide placed into a giant Pixy Stix. His father, Ronald Clark O'Bryan, was convicted of Tim's murder plus four counts of attempted murder. O'Bryan put potassium cyanide into five giant Pixy Stix that he gave to his son and daughter along with three other children. Only Timothy ate the poisoned candy and died.Bruce Nickell (5 June 1986) Murdered by his wife who poisoned a bottle of Excedrin.Richard Kuklinski (1935–2006)

Janet Overton (1942–1988) Her husband, Richard Overton, was convicted of poisoning her, but Janet's symptoms did not match those of classic cyanide poisoning, the timeline was inconsistent with cyanide poisoning, and the amount found was just a trace. The diagnostic method used was prone to false positives. Richard Overton died in prison in 2009.Urooj Khan (1966–2012), won the lottery and was found dead a few days later. A blood diagnostic reported a lethal level of cyanide in his blood, but the body did not display any classic symptoms of cyanide poisoning, and no link to cyanide could be found in Urooj's social circle. The diagnostic method used was the Conway diffusion method, prone to false positives with artifacts of heart attack and kidney failure.Autumn Marie Klein (20 April 2013), a prominent 41-year-old neuroscientist and physician, died from cyanide poisoning. Klein's husband, Robert J. Ferrante, also a prominent neuroscientist who used cyanide in his research, was convicted of murder and sentenced to life in prison for her death. Robert Ferrante is appealing his conviction. Mirna Salihin died in hospital on 6 January 2016, after drinking a Vietnamese iced coffee at a cafe in a shopping mall in Jakarta. Police reports claim that cyanide poisoning was the most likely cause of her death.

Jolly Thomas of Kozhikode, Kerala, India, was arrested in 2019 for the murder of 6 family members. Murders took place over a 14-year period, and each victim ate a meal prepared by the killer. The murders were allegedly motivated by wanting control of the family finances and property. Mei Xiang Li of Brooklyn, NY, collapsed and died in April 2017, with cyanide later reported to be in her blood. However, Mei never exhibited symptoms of cyanide poisoning and no link to cyanide could be found in her life.

Sararath "Am" Rangsiwutthiporn, who became quickly known as "Am Cyanide" in Thai media, was arrested by the Thai police for allegedly poisoning 11 of her friends and acquaintances, spanning 2020 to 2023, with 10 deaths and 1 surviving supposed victim. According to an ongoing investigation, the number of victims is currently at 20-30 persons, mostly dead with several survived.

In 1988, between 3,200 and 5,000 people died in the Halabja massacre owing to unknown chemical nerve agents. Hydrogen cyanide gas was strongly suspected. In 1995, a device was discovered in a restroom in the Kayabacho Tokyo subway station, consisting of bags of sodium cyanide and sulfuric acid with a remote controlled motor to rupture them, in what was believed to be an attempt by the Aum Shinrikyo cult to produce toxic amounts of hydrogen cyanide gas. In 2003, Al Qaeda reportedly planned to release cyanide gas into the New York City Subway system. The attack was supposedly aborted because there would not be enough casualties.

The lethal doses for NaCN reported for human adults vary with the type of exposure as follows: One to three mg/kg body weight if ingested; One hundred to 300 ppm if inhaled; and. One hundred mg/kg of body weight if absorbed.

H.I.3 COUNTER ATTACKS

350 Mg mass scale doses for population targets. Over 20 million doses planned for Canadian targets to stock

The average lethal dose for potassium cyanide is about 250 mg

Covert storage for access in Corporate drafting over denied trials 1990's-2023

Acute cyanide Intoxication: A rare case of survival

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4090999/

Flesh eating disease. Cyanide & Napalm will be administered covertly. Micro napalm to explode limbs, hands & internal organs with poisonings 

CANADA 2025-2026

5000 Canadians will be alive & free

The majority of just over 40 million will be replaced by look-a-likes & killed or wirelessly enslaved over H.I.3 2012-2023 rape-torture-terror attacks & prior efforts

Clean kills. Planned outbreaks & mass food-water supply tampering 

Cyanide compound paste concentrated 

Gloved clad drugging using Gravol sleep aid or Benedryl then cyanide paste fed forcefully through liquid toxification (paste to liquid)

Fruit punch. Soda options. Orange tang 

Overdose deaths... suicide 

You go to sleep & not wake up or a quick dizzy spell & you fall asleep & don't wake up

Tainting illegal street drugs causes overdoses leaving minimal users  

Lethal dart guns + diliberate overdose & dart retrieval. Manufactured or 3D print 

Screening humans for:

Credentials + career portfolio 

Earning potential 

... those falling short 

Overdoses (drug problems are rampant)

--------------------------------------------------------------------

Rothschild - Bennett